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7-TM GPCRs and Human Disease: Struggling between Regulation and Out of Regulation
Speaker:
裴刚 院士
中科院上海生命科学院院长
时间：12月14日 星期四 3-4pm
地点：北京大学 新生命科学楼 一楼大报告厅 （101 邓佑才报告厅）
Publications:
Activation of β-adrenergic receptor stimulates secretase activity and accelerates amyloid plaque formation. Nature Medicine (2006) 12:1390-6
β-Arrestins negatively regulate Toll-like/interleukin 1-receptor signaling through their functional interaction with TRAF6. Nature Immunology (2006) 7:139-47.
β-Arrestin2 functions as a phosphorylation-regulated suppressor of UV-induced NF-kappaB activation. EMBO J. (2005) 24:4237-46.
A Nuclear Function of beta-Arrestin1 in GPCR Signaling: Regulation of Histone Acetylation and Gene Transcription. Cell. (2005) 123:833-47.
Interaction with vesicle luminal protachykinin regulates surface expression of delta-opioid receptors and opioid analgesia. (2005) Cell 122:619-31.
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